The complex immunopathogenic mechanisms driving psoriasis involve interactions between keratinocytes and immune cells, overloading the cycle of inflammation that keeps patients plugged into their psoriasis - bound by skin plaques causing clinical symptoms of pain, itching, and burning sensations. 1,2...
As we shed light on the intricacies of psoriasis pathogenesis, we now know that other proinflammatory cytokines supplement the IL-17/23 axis and charge the immune cells in circulation. 2,3...
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